Liver and fat enzymes {11 beta HSD-1} can activate cortisol and make more triglycerides.
Fat cell hormones {adiponectin} can affect insulin and lipid metabolism.
Receptors {cannabis receptor} {CB1 receptor} that bind cannabis can stimulate appetite.
Liver proteins {FGF21 protein} can metabolize fat.
Gut peptides {ghrelin} can stimulate arcuate-nucleus appetite region.
Two hypothalamus peptides {orexin} {hypocretin}| can come from preprohypocretin and bind to lateral-hypothalamus receptors. They increase appetite and cause arousal. Hypocretin mutations can cause mammalian narcolepsy. Normal hypocretin is in Golgi organs, and mutated hypocretin is in smooth endoplasmic reticulum.
Fat-cell molecules {leptin}| can bind to hypothalamus receptors and suppress appetite. Leptin decreases arousal. Leptin stimulates satiation region and inhibits arcuate-nucleus appetite region.
Hormones {obestatin} can suppress appetite.
Fat-cell hormones {retinol-binding protein 4} can inhibit insulin receptors.
Cells release proteins {uncoupling protein 1} to ask for energy. Stimulating beta3-adrenergic and PPAR-nuclear receptors increases uncoupling protein 1 release.
4-Zoology-Organ-Endocrine Gland-Hormone
Outline of Knowledge Database Home Page
Description of Outline of Knowledge Database
Date Modified: 2022.0225